I think we can all agree that v1 of SARS CoV-2 is gone?
https://nextstrain.org/ncov/gisaid/global . Minus a small number of cases, it isn't circulating. So what are we chasing now and are the vaccines aimed at the right target? No, they aren't.
Boosters and new age group vaccine participants do not make any sense at all. The vaccines have not changed - the virus has. I know many TLDR, but read this article.
Quote:
In August, the companies began a trial of a multivalent vaccine that targets both the Delta and Alpha variants.
"We're not doing that because we actually think we need a new vaccine for those strains," says Philip Dormitzer, vice-president and chief scientific officer of viral vaccines and mRNA at Pfizer, based in New York City. "We want to practise all aspects of executing a strain change the preclinical research, the manufacturing, the clinical testing and the regulatory submissions so that if we do see a variant out there that truly escapes vaccine immunity, we're ready to go fast." Dormitzer says Pfizer currently has no plans to deploy its Beta or Delta vaccines among the public.
COVID vaccine makers brace for a variant worse than DeltaThis article should REALLY concern everyone. There is no new vaccine for what is out there...NOW. A 10/20 article - they are broadcasting what is coming. It's a historically observable outcome of leaky vaccines.
There are serious consequences to imperfect "vaccines" that are missing the broad array of clinical tests, deployed at the height of a pandemic. The evolutionary pressures a leaky vaccine place on a virus have been known for a very long time.
This has been posted before, but it worth posting again.
Marek's DiseaseQuote:
Because vaccination does not prevent infection with the virus, Marek's is still transmissible from vaccinated flocks to other birds, including the wild bird population. The first Marek's disease vaccine was introduced in 1970. The disease would cause mild paralysis, with the only identifiable lesions being in neural tissue. Mortality of chickens infected with Marek's disease was quite low. Current strains of Marek virus, decades after the first vaccine was introduced, cause lymphoma formation throughout the chicken's body and mortality rates have reached 100% in unvaccinated chickens. The Marek's disease vaccine is a "leaky vaccine", which means that only the symptoms of the disease are prevented. Infection of the host and the transmission of the virus are not inhibited by the vaccine. This contrasts with most other vaccines, where infection of the host is prevented. Under normal conditions, highly virulent strains of the virus are not selected. A highly virulent strain would kill the host before the virus would have an opportunity to transmit to other potential hosts and replicate. Thus, less virulent strains are selected. These strains are virulent enough to induce symptoms but not enough to kill the host, allowing further transmission. However, the leaky vaccine changes this evolutionary pressure and permits the evolution of highly virulent strains. The vaccine's inability to prevent infection and transmission allows the spread of highly virulent strains among vaccinated chickens. The fitness of the more virulent strains is increased by the vaccine.
The evolution of Marek's disease due to vaccination has had a profound effect on the poultry industry. All chickens across the globe are now vaccinated against Marek's disease (birds hatched in private flocks for laying or exhibition are rarely vaccinated). Highly virulent strains have been selected to the point that any chicken that is unvaccinated will die if infected.
Buy stock in Moderna, Pfizer and J&J if you haven't.
More about Marek, but what if the same scenario played out in human vaccines. t.u. receives a shout out - great for them, with their system ties to Wuhan it is fitting.
This chicken vaccine makes its virus more dangerousQuote:
"Our concern here, primarily and foremost, is whether this is going to happen with any of the vaccines that we give to people," said molecular biology of the University of Texas Austin, who specializes in the evolution of viruses and bacteria. "But there is a lot we don't know about how the scenario with Marek's could apply to newer human vaccines."
To test the imperfect vaccine hypothesis in humans, you would need monitor the vaccine response for either a large or isolated population for a long time. Doing this would allow a researcher to gauge how the vaccine interacts with the virus and if that relationship is evolving. Does the vaccine merely reduce symptoms, or does it also keep patients from getting infected and transmitting the virus?
We know the answer to the question. So what's the next move?
Bay fan asked, "Why do any of you care what others choose to do?"
Did I answer it for you?