Cibalo said:
My uncle got flesh eating bacteria and was never near any water. He opened an amazon box and got a paper cut on his finger that afternoon. That evening he went to a christmas party, came home and when he woke up in the morning his hand was swollen and painful.
HE went to the clinic and they rushed him to the hospital. Performed 3 surgeries; 1 to open up the wound and get as much of the infection out, 2 to install a central line for meds, and 3 to start cutting way dead tissue.
They found that putting him in for long periods of time in a hyperbarric chamber such that his blood became super saturated with oxygen greatly stopped the spread of the infection.
He went from the doctor saying they might take the whole arm, to below the elbow, to taking his ring and 2/3 of of the middle finger.
He was in the hospital for 3 weeks and then had to have a skin graf and rehab on his hand.
Vibrio vulnificus is not the only bacteria that causes necrotizing fasciitis (aka flesh eating diease). Multiple types of bacteria can cause it and you do not have to be exposed to water of any kind to contract many of these types of bacteria.
The following is an excerpt from the UpToDate online article on necrotizing fasciitis (
See the bolded portion regarding Vibrio vulnificus):
"Necrotizing fasciitis may be divided into two microbiologic categories: polymicrobial (type I) and monomicrobial infection (type II) :
Polymicrobial (type I) necrotizing infection is caused by aerobic and anaerobic bacteria.
Typically, at least one anaerobic species (most commonly
Bacteroides,
Clostridium, or
Peptostreptococcus) is isolated in combination with Enterobacteriaceae (eg,
Escherichia coli,
Enterobacter,
Klebsiella,
Proteus) and one or more facultative anaerobic streptococci (other than group A
Streptococcus [GAS]). Obligate aerobes (such as
Pseudomonas aeruginosa) are rarely components of such mixed infections. Uncommonly, fungi (predominately
Candida species) are recovered in polymicrobial (type I) necrotizing infection.
Fournier gangrene is caused by facultative organisms (
E. coli,
Klebsiella, enterococci) along with anaerobes (
Bacteroides,
Fusobacterium,
Clostridium, anaerobic or microaerophilic streptococci). (See
'Involved sites' below.)
Necrotizing infection of the head and neck is usually caused by mouth anaerobes (such as Fusobacteria, anaerobic streptococci,
Bacteroides, and spirochetes). (See
'Involved sites' below.)
Other terms for polymicrobial (type I) infection include synergistic necrotizing cellulitis and progressive bacterial synergistic gangrene.
Monomicrobial (type II) necrotizing infection is usually caused by GAS or other beta-hemolytic streptococci. Infection may also occur as a result of
Staphylococcus aureus. Infection with no clear portal of entry occurs in about half of cases; in such circumstances, the pathogenesis of infection likely consists of hematogenous translocation of GAS from the throat (asymptomatic or symptomatic pharyngitis) to a site of blunt trauma or muscle strain.
M protein is an important virulence determinant of GAS. Necrotizing infection caused by GAS strains with M types 1 and 3 is associated with streptococcal toxic shock syndrome in about 50 percent of cases. GAS strains of these and other serotypes can produce pyrogenic exotoxins, which induce cytokine production, likely contributing to shock, tissue destruction, and organ failure. (See
"Group A streptococcus: Virulence factors and pathogenic mechanisms" and
"Invasive group A streptococcal infection and toxic shock syndrome: Epidemiology, clinical manifestations, and diagnosis".)
Less frequent causes of monomicrobial (type II) necrotizing infection include Vibrio vulnificus and Aeromonas hydrophila. Infections due to these pathogens typically occur in the setting of traumatic injury associated with sea water or fresh water, respectively. Other risk factors for necrotizing infection due to V. vulnificus include cirrhosis and ingestion of contaminated oysters. (See "Vibrio vulnificus infections".)The incidence of necrotizing fasciitis ranges from 0.3 to 15 cases per 100,000 population:
Polymicrobial (type I) necrotizing fasciitis (caused by aerobic and anaerobic bacteria) usually occurs in older adults and/or in individuals with underlying comorbidities. The most important predisposing factor is diabetes, especially with associated peripheral vascular disease. (See
'Risk factors' below.)
Monomicrobial (type II) necrotizing fasciitis (most commonly caused by GAS) may occur in any age group and in individuals with no underlying comorbidities. In the United States, there are an estimated 3.5 cases of invasive GAS infections per 100,000 persons; necrotizing infections make up approximately 6 percent of these cases.
Necrotizing myositis Necrotizing myositis is an infection of skeletal muscle typically caused by group A
Streptococcus (and other beta-hemolytic streptococci). It may be preceded by skin abrasions, blunt trauma, or heavy exercise. Necrotizing myositis is rare. One report noted 21 cases documented between 1900 and 1985; another review of over 20,000 autopsies noted 4 cases.
Clostridial myonecrosis (gas gangrene) is discussed separately. (See
"Clostridial myonecrosis".)
Necrotizing cellulitis Necrotizing cellulitis is typically caused by anaerobic pathogens and may be divided into two types: clostridial (usually caused by
Clostridium perfringens; less frequently
Clostridium septicum) and nonclostridial (caused by polymicrobial infection).
In both types, crepitus is observed in the skin, but there is sparing of the fascia and deep muscles. Pain, swelling, and systemic toxicity are not prominent features, and the relative mildness helps distinguish cellulitis from clostridial myonecrosis (gas gangrene). (See
"Clostridial myonecrosis".)"
Source (It is from a pay site and you have to have a subscription to access):
https://www.uptodate.com/contents/necrotizing-soft-tissue-infections?search=necrotizing%20fasciitis%20causes§ionRank=1&usage_type=default&anchor=H650406663&source=machineLearning&selectedTitle=1~140&display_rank=1#H650406663 CM
No material on this site is intended to be a substitute for professional medical advice, diagnosis or treatment. See full
Medical Disclaimer.